Synopsis:
In this work, we evaluated the concentration of the ATG7 protein and P62 protein and the β- glucocerebrosidase enzyme in the CSF of the patients with increased pulmonary encephalopathy. Atg7 and the ubiquitin-like conjugation it participated in, may be an important channel of the formation of autophagy in neurons of patients with pulmonary encephalopathy. Through this research, we can understand the molecular mechanisms of the formation of autophagy in neurons of patients with pulmonary encephalopathy, which may be new targets for diagnosis and treatment of pulmonary encephalopathy.
