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The Role of Neurous Autophagy in Pulmonary Encephalopathy and Signalling Pathways

DOI: 
10.7727/wimj.2015.208
Synopsis: 
In this work, we evaluated the concentration of the ATG7 protein and P62 protein and the β- glucocerebrosidase enzyme in the CSF of the patients with increased pulmonary encephalopathy. Atg7 and the ubiquitin-like conjugation it participated in, may be an important channel of the formation of autophagy in neurons of patients with pulmonary encephalopathy. Through this research, we can understand the molecular mechanisms of the formation of autophagy in neurons of patients with pulmonary encephalopathy, which may be new targets for diagnosis and treatment of pulmonary encephalopathy.

ABSTRACT

Objective: To survey the effects of hypoxia on autophagy in the pulmonary encephalopathy.

Accepted: 
27 Apr, 2015
PDF Attachment: 
Journal Sections: 
e-Published: 14 Oct, 2015

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Manuscripts that are Published Ahead of Print have been peer reviewed and accepted for publication by the Editorial Board of the West Indian Medical Journal. They may appear in their original format and may not be copy edited or formatted in the style guide of this Journal. While accepted manuscripts are not yet assigned a volume, issue or page numbers, they can be cited using the DOI and date of e-publication. See our Instructions for Authors on how to properly cite manuscripts at this stage. The contents of the manuscript may change before it is published in its final form. Manuscripts in this section will be removed once they have been issued to a volume and issue, but will still retain the DOI and date of e-publication.

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